Artificial Sweetner
An Artificial Sweetner is a sugar substitute that is not naturally occurring.
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- Example(s):
- Counter-Example(s):
- See: Siraitia Grosvenorii, Food Additive, Sweetness, Taste, Food Energy, Turbinado.
References
2018
- (Wikipedia, 2018) ⇒ https://en.wikipedia.org/wiki/sugar_substitute Retrieved:2018-4-23.
- A sugar substitute is a food additive that provides a sweet taste like that of sugar while containing significantly less food energy. Some sugar substitutes are produced by nature, and others produced synthetically. Those that are not produced by nature are, in general, called artificial sweeteners. In 2017, sucralose was the most common sugar substitute used in manufacturing of foods and beverages, having 30% of the global market projected to become $2.8 billion in collective value by 2021.
When sweeteners are provided for restaurant customers to add to beverages such as tea and coffee, they are often available in paper packets that can be torn and emptied. In North America, the colors are typically white for sucrose, blue for aspartame, pink for saccharin,yellow for sucralose (United States) or cyclamate (Canada), tan for turbinado, orange for monk fruit extract, and green for stevia.
- A sugar substitute is a food additive that provides a sweet taste like that of sugar while containing significantly less food energy. Some sugar substitutes are produced by nature, and others produced synthetically. Those that are not produced by nature are, in general, called artificial sweeteners. In 2017, sucralose was the most common sugar substitute used in manufacturing of foods and beverages, having 30% of the global market projected to become $2.8 billion in collective value by 2021.
2015
- http://sugarscience.ucsf.edu/artificial-sweeteners-induce-glucose-intolerance.html
- QUOTE: … A team of scientists found that artificial sweeteners change the huge colony of bacteria in your intestines to favor the harmful bacteria that increase risk for diabetes. Scientists fed 10-week-old mice drinks containing either: saccharin (the sweetener in the pink packets of Sweet’N Low), sucralose (the yellow packets of Splenda), aspartame (the blue packets of Equal), sugar, plain water.
The amount given to the mice was equivalent to four diet sodas per day for a human. After one week, the mice fed diet drinks had higher than normal blood sugar levels in response to eating sugar, while those that drank sugared water or plain water did not. Saccharin, aspartame and sucralose are chemically very different from each other, but they all caused the same abnormal rise in blood sugar. A high rise in blood sugar after eating sugar is called “glucose intolerance”, which is a marker for diabetes and increased risk for heart attacks.
- QUOTE: … A team of scientists found that artificial sweeteners change the huge colony of bacteria in your intestines to favor the harmful bacteria that increase risk for diabetes. Scientists fed 10-week-old mice drinks containing either: saccharin (the sweetener in the pink packets of Sweet’N Low), sucralose (the yellow packets of Splenda), aspartame (the blue packets of Equal), sugar, plain water.
2014
- (Suez et al., 2014) ⇒ Jotham Suez, Tal Korem, David Zeevi, Gili Zilberman-Schapira, Christoph A. Thaiss, Ori Maza, David Israeli et al. (2014). “Artificial Sweeteners Induce Glucose Intolerance by Altering the Gut Microbiota.” Nature 514, no . 7521
- ABSTRACT: Non-caloric artificial sweeteners (NAS) are among the most widely used food additives worldwide, regularly consumed by lean and obese individuals alike. NAS consumption is considered safe and beneficial owing to their low caloric content, yet supporting scientific data remain sparse and controversial. Here we demonstrate that consumption of commonly used NAS formulations drives the development of glucose intolerance through induction of compositional and functional alterations to the intestinal microbiota. These NAS-mediated deleterious metabolic effects are abrogated by antibiotic treatment, and are fully transferrable to germ-free mice upon faecal transplantation of microbiota configurations from NAS-consuming mice, or of microbiota anaerobically incubated in the presence of NAS. We identify NAS-altered microbial metabolic pathways that are linked to host susceptibility to metabolic disease, and demonstrate similar NAS-induced dysbiosis and glucose intolerance in healthy human subjects. Collectively, our results link NAS consumption, dysbiosis and metabolic abnormalities, thereby calling for a reassessment of massive NAS usage.